The contamination of organochlorine PESTICIDES (OCPs) from the selected rivers in Okinawa Island was investigated to estimate the current status of pollution in water, plants and surface sediments in these rivers. The Aja River, Asato River, Houtoku River, Kokuba River and Okukubi River were selected for this study. The concentration of the total PESTICIDES were in the range of 0.94-231.8 ng/L in river water, 0.006-191.6 ng/L dry weight in river sediments and 0.001-55.8 ng/L dry weight in plants. Among the OCPs, ΣHCB of α-BHC,β-BHC and Aldrin were the common detected compounds in river water. The α-BHC, Aldrin and Dieldrin were the most frequent detected compounds in river sediments and α-BHC and Dieldrin were the common OCPs detected in plants. Aldrin, Dieldrin and ΣHCB were in abundance. Various contamination patterns between the selected river water, sediments and plants were observed. Aja River, Asato River and Houtoku River were contaminated with α,β,δ,γ- BHC,  Aldrin and Dieldrin (water and sediments) whereas the main OCPs in Kokuba River and Okukubi River were Dieldrin (plants) The OCPs levels in all rivers were generally below guideline values in Japan, but some sites displayed levels which exceeded the EC and WHO Standards for Aldrin, Dieldrin and α, β-BHC.

Exposure to PESTICIDES could be one of the contributing cause to the falling sperm counts and rising levels of male infertility.Adverse effects of PESTICIDES in the environment first received widespread attention in the 1960s. However, only recently it has been postulated that long term, low exposure of these chemicals are increasingly linked to human health effects such as immuno-suppression, endocrine disruption, reproductive abnormalities and cancer. This article critically reviewed the epidemiological studies of reproductive toxicity of different PESTICIDES in males. According to previous studies it is shown that exposure to PESTICIDES was significantly associated with sperm levels well below the limit for male fertility. No large-scale studies assessing pesticide exposure and its relationship to infertility have been done. To overcome the difficulties in interpretation and to reach strong conclusions, future studies on human male reproductive effects of different types of PESTICIDES should consider several methodological problems. So, we conclude integrated studies considering many factors are warranted to draw definite conclusion and also in the view of adverse health effects observed to some extent in workers with few PESTICIDES, it is necessary now to find out ecologically sound alternatives to PESTICIDES and also to educate the workers/farmers about the safe use of these PESTICIDES to reduce reproductive health risk associated with exposure.