Introduction: Eye trauma is a common cause of one-sided blindness in children and teenagers. Blunt trauma can cause rupture of orbital cavity and the herniation of its content may affect the nearby sinuses leading to hemorrhage in the anterior chamber (hyphema).Traumatic hyphema that specially happens in the eyes with blunt trauma can be followed by high infra ocular pressure, corneal staining, cataract, injury of the optic nerve and sever LOSS of visual acuity (VA). Early diagnosis and treatment of this complication can prevent the later and worst complications. The aim of this study is to determine the prevalence of traumatic hyphema and its complications in eye blunt trauma.Materials and Methods: According to a cross- sectional descriptive protocol, we studied 83 patients with eye blunt trauma who referred to the eye clinics of Bahman 22nd and Farabi hospitals. A formal questionnaire was designed to be completed for personal particularities of each patient as well as the information about the examinations of orbit, cornea, AC, lens and optic nerves. In addition, the Intraocular Pressure (IOP) and initial VA were recorded. Then we analyzed these records and evaluated the prevalence of hyphema and its complications in eye blunt trauma.Results: This study shows that hyphema is a common complication of eye blunt trauma that occurs in approximately 50% of the patients with eye blunt trauma.36.1% of the patients were in the age group of 20-40 years old with an average age of 36.7 years. There were 65.9% males and 34.1% females. The most common causes of trauma included projectiles (48.8%) and assaults (36.6%). The most common associated symptoms with hyphema included red eye (56.09%) and dark VISION (48.7%).The most frequent occurring complications included sever decrease of initial VA (75.6%) and high IOP (48.8%).Conclusion: Hyphema is a common complication of eye blunt trauma that may lead to other complications like sever LOSS of VA and high IOP. So, thinking of and searching for hyphema in any patient with eye blunt trauma can help us to diagnose it at early stages and prevent other complications.

Dear Editor, VISION LOSS is rarely associated with noninfectious human immunodeficiency virus (HIV) retinopathy in HIV positive patients with normal CD4 counts. Ophthalmologists should be aware that HIV retinopathy may be the cause of visual LOSS in a subset of HIV patients without infectious neuroretinitis who are otherwise stable. Electrophysiological testing has high diagnostic value in these patients.

Objective: This research was conducted in order to comparing some memory components in Students with and without VISION LOSS. Method: The sample consisted of 38 students aging 16 to 20, studying in Ardabil high schools. Utilizing Wechsler memory scale, mental control, logical memory, reversed repetition, and word association were evaluated. Results: MANOVA analysis revealed that students with VISION LOSS had better abilities in mental control, logical memory, and word association subtests. No significant difference was observed for reverse repetition. Conclusion: We can probably conclude that students with VISION LOSS show good capabilities on tasks requiring listening skills and attention.

Purpose: To report a rare case of Woakes’,syndrome presented with bilateral VISION LOSS. Methods: A 28-year-old male with a 1-year history of VISION LOSS in the left eye was referred to the neuro-ophthalmology clinic after sudden VISION LOSS in his right eye. A detailed review of clinical findings and the presumed pathophysiological basis of VISION LOSS was performed. Results: Neuroimaging revealed bilateral massive nasal polyps, sphenoid sinus mucocele formation, and optic nerve dehiscence inside the sphenoid sinus. The VISION in the right eye was restored after pulse corticosteroid therapy,however, the left eye remained severely visually compromised even after nasal polypectomy and mucocele drainage. Conclusion: Sinonasal disorders should be sought for patients with unexplained VISION LOSS, as prompt intervention could be VISION-saving in these patients.

Purpose: To report visual and anatomical outcome of flap LOSS after LASIK surgery for correction of high myopia. Materials & Methods: Retrospective evaluation of flap LOSS in two eyes of two patients, a rare complication of LASIK Surgery. Case No. 1: 27 y/o woman with Ref. OD -9.75; 2.75*177 VA. CC: 20/60 KR OD: 41.86*77 39.86 * 168 & partial amblyopia due to anisometropia. Case No. 2: 38 y/o woman with Ref OD. -9.50 1.0*177 VA CC: 20/30 KR OD: 42.06*77 40.99 * 167. Results: In both patients during LASIK surgery free cap complication occurred and after Laser therapy free cap positioned over the stromal bed, but the next day after operation flaps were lost. For case NO.1only medical management performed and after 4.5 years F/U cornea has 2° central haziness. For case NO.2 another corneal flap from donor globe cut and sutured with Nylon 10/0 and after 18 months F/U cornea is clear. Conclusion: In patients with flat keratometry less than 41.0 Diopter rate of free cap are high and after laser therapy for high myopia the keratometry became more flat. If surgeon doesn't suture flap the rate of flap LOSS increase so for prevent of corneal haziness and corneal ectazia this evaluation suppose to perform donor corneal flap graft.      

Neurological manifestation may complicate Takayasu arteritis (TA). A 23-year-old girl with sudden onset of VISION LOSS was admitted to hospital. Her brain MRI showed abnormal T2-signal hyperintensity and visual evoked potential revealed prolonged P100 latency. Consequently, optic neuritis was diagnosed. A review on history of dizziness, falling, and weak pulses of upper extremities led to more investigation. Angiography revealed a total occlusion of right and left carotids, left vertebral arteries, aneurismal dilatation of innominate artery and critical stenosis of right vertebral artery. Following diagnosis of TA, stenting of right vertebral artery was done, but she passed away because of subarachnoid hemorrhage.