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Paper Information

Journal:   ARCHIVES OF RAZI INSTITUTE   June 2003 , Volume - , Number 55; Page(s) 111 To 116.
 
Paper: 

CASE REPORT OF ENCEPHALOPATHY IN GOATS

 
 
Author(s):  GHOLAMI M.R.*, HABL ALVARID M.H., BAZARGANI T., EZI A.
 
* Pathology Dept., Razi Vaccine & Serum Research Institute, Tehran, Iran
 
Abstract: 

Polioencephalomalacia is none infectious neuro degenerative disease that affect sheep, goat, deer and cow. Polioencephalomalacia commonly occur in cattle fed ration rich in carbohydrates with little roughage that may changes microflora of rumen and impaired synthesize of thiamine (Thomson et al 2001, Boyd & Walton 1977). Polioencephalomalacia could represent a multifactoral metabolic disorder with multiple etiologies such as nutritional problem, high level of carbohydrate, water deprivation, salt intoxication, high level intake of sulfur, renal encephalopathy, amprolium and some plant such as Bracken fern and Horsetail. Probable mechanisms that may cause thiamine deficiency polioencephalomalacia in ruminant are disorder of absorption, synthesize and destruction of thiamine by thiaminase (Radostit et al 1994, Bakker et al 1980, Jeffrey et al 1994, Loew & Dunlop 1972, Olkowski 1992, Thomber et al 1979). Evidence (or theories) linking thiamine with the ruminant Polioencephalomalacia disorder include clinical response to thiamine injection in some individual (Thomson el al 2001).

 
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